Sympathetic Nervous System Neuroplasticity

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Sympathetic nervous system neuroplasticity.

In human and experimental models of heart failure there is widespread change in the sympathetic nervous system. This involves preferential activation of the sympathetic nervous outflow to the heart, impairment of norepinephrine reuptake by sympathetic nerves, and reduction of the content of norepinephrine in the failing myocardium, attributable in part to sympathetic neuronal rarefaction.1–4 Mi...

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Sympathetic Nervous System and Hypertension Print ISSN: 0194-911X. Online ISSN: 1524-4563 Copyright © 2013 American Heart Association, Inc. All rights reserved. is published by the American Heart Association, 7272 Greenville Avenue, Dallas, TX 75231 Hypertension doi: 10.1161/HYPERTENSIONAHA.111.00633 2013;61:556-560; originally published online January 28, 2013; Hypertension. http://hyper.ahajo...

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We evaluated sympathetic nervous system activity by sympathetic skin response (SSR) recording and we further investigated sympathetic and opioid outflow indirectly in patients with features of reflex sympathetic dystrophy by measuring concentrations of plasma catecholamines (CAs) and their metabolites and plasma metenkephalin (ME), before and after corticoid treatment. Six patients were studied...

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Sympathetic nervous system and deoxycorticosterone-saline hypertension.

We wish to comment on the article by de Champlain and Ameringen (Circ Res 31:617-628, 1972) in which they implicate the involvement of the peripheral sympathetic nervous system in the etiology of deoxycorticosterone (DOCA)-saline hypertension. In the Discussion of their article, the work of Clarke et al. (Life Sci [I] 9:1097-1108, 1970) has been misquoted. Contrary to the authors' impression, w...

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The sympathetic nervous system and heart failure.

Heart failure (HF) is a syndrome characterized by upregulation of the sympathetic nervous system and abnormal responsiveness of the parasympathetic nervous system. Studies in the 1980s and 1990s demonstrated that inhibition of the renin-angiotensin-aldosterone system with angiotensin-converting enzyme inhibitors improved symptoms and mortality in HF resulting from systolic dysfunction, thus pro...

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ژورنال

عنوان ژورنال: Hypertension

سال: 2006

ISSN: 0194-911X,1524-4563

DOI: 10.1161/01.hyp.0000200024.12746.f3